| Mélatonine : régulation du sommeil |
Protection du système hormonal

Melatonin
It has anti-radical, anti-tumor, and sleep inducing properties. At high doses, it also possesses anti-epileptic properties.
The reduction in melatonin observed in many studies could therefore cause a decrease in the recycling systems of free radicals with, as a consequence, the acceleration of oxidative stress, premature aging, and acceleration of an already developing tumor process.
Sleep disorders are also possible, causing in turn, a cascade of tiredness and fatigability that can lead to depression..
A significant reduction in melatonin could also lead to an increase in epileptic seizures in patients or predisposed individuals.
Young chickens - exposed 38 days to VDU screens –computer and TV - (cathode ray tubes) (Bastide M, 1997 - Youbicier-Simo B-J ,2001 Univ. Montpellier)
| Anticorps : défenses du corps pour contrer toute molécule étrangère |
Protection du système immunitaire
 
Immune response
Immune depression in young chicks exposed to screens should lead to work focused on the possibility of benign chronic or recurring infections like a “head cold”, or even more serious ones, as factors contributing to further compromising HIV carriers or other slow developing virus.
Simultaneously to hormonal depletions, the production of IgG anti-bodies (immunoglobuline G: a protein of the class of antibodies) is suppressed amongst chickens which are exposed to a cathode ray tube screen, as well as the number of white blood corpuscles (monocytes) decreases, which shows a depression of the immune system. Those that are exposed + CMO show a level of antibodies and a production of monocytes that equals the level shown by those that are not exposed.
IgG : Young chickens - exposed 38 days to VDU screens (cathode ray tubes) (Bastide M, 1997 - Youbicier-Simo B-J ,2001 Univ. Montpellier
Monocytes : Mice - exposed 15 weeks to VDU screens (cathode ray tubes) (Dr. Faivre-Bonhomme ; Hôpital Paul Brousse, Paris)
| ACTH : mesure du stress biologique |
Protection du système hormonal

Changes in hormone release can also be a primary consequence of these ionic abnormalities, with abnormal changes in ACTH, cortico and mineral-steroid hormones, calcitonin, PTH, and melatonin.
(B.J. Youbicier-Simo, M. Bastide, -Montpellier University, 1997, 2001. Alonso G.-CNRS Montpellier; Dayanithi G, -INSERM Montpellier, 2001)
Changes in these hormones cause, in turn, systemic disorders:
- In the nervous and muscular systems , psychological instability, irritability, tendency towards depression, concentration and memory disorders, insomnia, muscular weakness, cramps, contractions.
- In the immune system, a drop in the performance of defense mechanisms against bacteria, virus, parasites, allergies ( degranulation facilitation of histamine releasing cells), tendency towards maintenance or worsening of inflammatory or auto-immune diseases in genetically predisposed individuals.
Mice – exposed to cellular phones GSM / 15 weeks (Dayanithi G – INSERM, Univ Montpellier
| Calcium : troubles hormono-immunitaires et nerveux |
Protection des échanges ioniques cellulaires

Calcium ions are involved in the activity of numerous body enzymes. Subsequently, any change in the intracellular level of calcium is likely to induce changes in intracellular enzymes, notably those of kinase proteins involved in signal transduction. (Dayanithi G, -INSERM Montpellier)
For example, in rat pituitary cells exposed to electromagnetic radiation from a mobile phone, the reported simultaneous increase in the intracellular concentration of Ca++ and the increased secretion of ACTH (adrenocorticotropic hormone) have modified the activity of enzymes involved in the synthetic cascade of this hormone.
When there is an accumulation of intracellular calcium, the latter is the result of a shift in ion charges, which are otherwise deficient in other parts of the body. It is possible to assume that these changes in intra and extracellular concentrations cause the body to make use of mechanisms designed to recover or return calcium to “normal” levels, inducing, as such, cellular stress with a risk of a loss in balance during long term exposure, or the striking of a new balance/equilibrium on another potentially pathological level of functioning.
Ionic movement of Ca++ is subsequently going to have an impact on the movement of other ions whose metabolism is interdependent, notably inverse shifts in Mg++ (Magnesium).
In terms of the Central Nervous System (CNS) and the neuromuscular system, it is known that Ca++ plays a very important role in neuromuscular excitability, notably in the autonomous nervous system. These abnormalities could result in disorders like spasmophilia with other kinds of malaise and functional secondary deregulation of organs or main functions such as the heart, circulation, digestion, respiration, pain sensitivity or sensitivity to other stimuli.
Mice – exposed to cellular phones GSM / 15 weeks (Dayanithi G – INSERM, Univ Montpellier
| Cortisol, corticostérone : gestion du stress, régulations hormonales |
Protection du système hormonal
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Simultaneously to hormonal depletions, the production of IgG anti-bodies (immunoglobuline G: a protein of the class of antibodies) is suppressed amongst chickens which are exposed to a cathode ray tube screen, as well as the number of white blood corpuscles (monocytes) decreases, which shows a depression of the immune system. Those that are exposed + CMO show a level of antibodies and a production of monocytes that equals the level shown by those that are not exposed.
IgG : Young chickens - exposed 38 days to VDU screens (cathode ray tubes) ; (Bastide M, 1997 - Youbicier-Simo B-J ,2001 Univ. Montpellier)
Monocytes : Souris exposées 15 semaines à un écran informatique cathodique ; (Dr. Faivre-Bonhomme ; Hôpital Paul Brousse, Paris)
| ACTH : mesure du stress biologique |
Protection du système hormonal
|
Augmentation de 400% de la libération de l'hormone de stress
par rapport aux témoins non exposés.
Normalisation avec l'oscillateur magnétique de compensation CMO
|
|

Corticosterone – ACTH
Changes in hormone release can also be a primary consequence of these ionic abnormalities, with abnormal changes in ACTH, cortico and mineral-steroid hormones, calcitonin, PTH, and melatonin.
Changes in these hormones cause, in turn, systemic disorders:
- In the nervous and muscular systems , psychological instability, irritability, tendency towards depression, concentration and memory disorders, insomnia, muscular weakness, cramps, contractions.
- In the immune system, a drop in the performance of defense mechanisms against bacteria, virus, parasites, allergies ( degranulation facilitation of histamine releasing cells), tendency towards maintenance or worsening of inflammatory or auto-immune diseases in genetically predisposed individuals.
Souris exposées tel. portable GSM durant 15 semaines. (Dayanithi G - Univ Montpellier).
| Calcium : troubles hormono-immunitaires et nerveux |
Protection des échanges ioniques cellulaires
Multiplication par 2 de la concentration en calcium
par rapport aux témoins non exposés.
Normalisation avec l'oscillateur magnétique de compensation CMO
|
|

Calcium
Calcium ions are involved in the activity of numerous body enzymes. Subsequently, any change in the intracellular level of calcium is likely to induce changes in intracellular enzymes, notably those of kinase proteins involved in signal transduction.
For example, in rat pituitary cells exposed to electromagnetic radiation from a mobile phone, the reported simultaneous increase in the intracellular concentration of Ca++ and the increased secretion ACTH (adrenocrticotropic hormone) have modified the activity of enzymes involved in the synthetic cascade of this hormone.
When there is an accumulation of intracellular calcium, thelatter is the result of a shift in ion charges, which are otherwise deficient in other parts of the body. It is possible to assume that these changes in intra and extracellular concentrations cause the body to make use of mechanisms designed to recover or return calcium to “normal” levels, inducing, as such, cellular stress with a risk of a loss in balance during long term exposure, or the striking of a new balance/equilibrium on another potentially pathological level of functioning.
Ionic movement of Ca++ is subsequently going to have an impact on the movement of other ions whose metabolism is interdependent, notably inverse shifts in Mg++ (Magnesium).
In terms of the Central Nervous System (CNS) and the neuromuscular system, it is known that Ca++ plays a very important role in neuromuscular excitability, notably in the autonomous nervous system. These abnormalities could result in disorders like spasmophilia with other kinds of malaise and functional secondary deregulation of organs or main functions such as the heart, circulation, digestion, respiration, pain sensitivity or sensitivity to other stimuli.
Souris exposées tel. portable GSM durant 15 semaines. (Dayanithi G - INSERM U432, Montpellier).
| Cortisol, corticostérone : gestion du stress, régulations hormonales |
Protection du système hormonal
Division par 2 de la production de
par rapport aux témoins non exposés.
Normalisation avec l'oscillateur magnétique de compensation CMO
|
|
 
Hormonal system
The production of corticosterone (in young chickens) and cortisol (in mice), both adrenal hormones, is stimulated by the pituitary gland (or hypophysis) in the brain, which is the “crossroads” of hormonal and immune regulations, themselves interdependent. This gland controls biological stress and counteracts inflammatory processes.
The state of biological stress observed in exposed animals returns to normal with the compensatory oscillator. These same results will be shown also in humans, precisely in the study of chronic stress symptoms.
The virtual suppression of the melatonin production under EMF exposure clearly shows the inability of exposed animals to control their stress levels. On the contrary, the presence of the compensatory oscillator enables this hormone to reach normal rates. For that matter, melatonine is well known for being electromagneto-sensitive.
Corticosterone : Young chickens - exposed 38 days to VDU screens –computer and TV - (cathode ray tubes) (Bastide M, 1997 - Youbicier-Simo B-J ,2001 Univ. Montpellier)
Cortisol : Health mice - exposed for 5 weeks to cell phone ( GSM ) (Youbicier-Simo B-J , Bastide M., 2001 Univ. Montpellier)
| Mortalité embryonnaire : mesure de la toxicité biologique |

The study of embryo mortality rate of a species (chickens) reveals abnormalities that appeared during its development and which led to its death.
This study shows that even for a continuous exposure to the polluting device radiation (leading to the death of the majority of embryos), the compensatory oscillation is capable of maintaining or protecting the fundamental vital processes, reducing the rate of mortality to almost the normal level.
On the contrary, these results highlight the level of severe biological toxicity of EMF from the apparatus that were tested and particularly that of ELFs. Indeed, the virtual elimination, via filtering, of micro-wave radiation from mobile phones did not reduce the level of toxicity (NB. The magnetic field of ELFs may not be filtered or suppressed).
Embryo mortality
The observance of a very significant rise in embryo mortality in chickens can result in considering the possibility of early spontaneous abortions, the increase of which has already been debated with screen work.
% Mortality of embryos (chickens) (Youbicier-Simo B-J., Bastide M., 1997-2001University of Montpellier)
| Micro-noyaux : signe possible d'un dysfonctionnement du cycle cellulaire |
Protection génétique

Cell cores
The multiplication of micro-cores observed in immune cells (lymphocytes – macrophages) is a sign of dysfunction of the cells in question resulting in their death and destruction or in their anarchic development leading to a subsequent rise in cancer risk.
This test consists of counting under microscope the number of fragments of DNA that are present and excessive in the peritoneum macrophages (white corpuscles, cells of the immune system) of animals that have been exposed or those that have been exposed + CMO.
These micronuclei that are present in the cell represent one initial possible step of cancerous process, if these abnormal cells are not eliminated by defence systems of the organism.
Therefore, the high number of cells that include several micronuclei (over 2 cores) for the GSM exposed organisms - clearly shows the impact of its radiation at the fundamental level of the biological system - and the virtual normalisation obtained with the compensatory oscillation (CMO) confirms the results in embryogenesis, as described above.
Mice – exposed to cell phone GSM / 15 weeks Youbicier-Simo B-J - Tecnolab ; Fernandez A.- CNRS, Univ.
| ADN : marqueurs de souffrance cellulaire |
Protection du système génétique

These results demonstrate cellular stress linked to the exposure to a mobile telephone, through increased production of the HSP 70 proteins (3.6 times base level), as well as the overproduction of the cell’s growth factor by the activation of SRE (multiplied by 3.7).
Following this study, the authors underline the necessity to question the « safety standards» applied to mobile telephones based on SAR (Specific Absorption Rate) by adding to them or replacing them by biological value that take into account the genetic response to this electromagnetic pollution. HSP provides viable and sensitive biological markers which can act as the basis for realistic safety standards in respect of mobile telephones.
The return to normal of SRE (at 100%) through CMO protection, together with the reduction of 73% in HSP, in comparison to the organisms exposed to radiation from a GSM telephone, provide an irrefutable biochemical proof of the effectiveness of this innovative biotechnology.
These results of protection of the stress proteins and DNA through the addition of compensating electromagnetic oscillators (CMO-Tecno AO) to mobile phones, corroborates the results of bioprotection for all the biological parameters of cellular and systemic stress previously studied with CMO (micronuclei, calcium, ACTH, corticosterone, neurogenesis, embryonic mortality, melatonin, cortisol, monocytes, nitric oxide)
This publication in the « Journal of Cellular Biochemistry » is a further validation of the CMO-Tecno AO technology, which brings a reply and a solution to the problem of biological effects caused by exposure to mobile telephones. The confirmation of the biological effectiveness of this protective technology at the level of the most critical controls of cells (DNA), should make it easy for Industrialists and Politicians to put into effect the Precautionary Principle.
| ADN : prolifération des neurones |
Protection sur la neurogénèse

Neurogenesis
The decrease in the neurogenesis of hippocampus cells could be at the root of memory disorders; since this structure is involved in short term memorization and learning mechanisms. A renewal problem for these cells could cause abnormalities of these functions.
Mice – exposed to cell phones GSM / 11 weeks Alonso G. - CNRS, Univ. Montpellier ; Youbicier-Simo B-J - Tecnolab
| ADN : prolifération cellulaire |
Protection du système génétique

Le SRE (Serum Response Element) est la séquence régulatrice de l'ADN.
Mesurer le niveau de SRE permet de quantifier la croissance cellulaire
These results demonstrate cellular stress linked to the exposure to a mobile telephone, through increased production of the HSP 70 proteins (3.6 times base level), as well as the overproduction of the cell’s growth factor by the activation of SRE (multiplied by 3.7).
The return to normal of SRE (at 100%) through CMO protection, together with the reduction of 73% in HSP, in comparison to the organisms exposed to radiation from a GSM telephone, provide an irrefutable biochemical proof of the effectiveness of this innovative biotechnology.
Mouches Drosophila melanogaster exposées tel. portable GSM durant les 10 jours de leur développement (de la ponte des oeufs aux larves).
(Goodman R - Dept of Pathology, Columbia University Health Sciences, New York).
|